Jesse Luke -Phunky_Pharm
@realjesseluke.bsky.social
Biomed writer, #Gonzo Neuropsychopharm, GABAA, PD/PK, PhunkyPharmacology.org , #psychsky #pharmacology #medsky Posts not medical advice.
Fixed link zenodo.org/records/1761...
The Algorithmic Psyche: An Information-Theoretic and Systemic Analysis of Risks in LLM-Powered Mental Healthcare
Beyond Bugs: 'Designed Failures' and the Systemic Risks of LLMs in Psychiatry Description: This paper challenges the prevailing safety paradigm of Large Language Models (LLMs) in mental healthcare by ...
zenodo.org
November 16, 2025 at 1:19 PM
Fixed link zenodo.org/records/1761...
For something of that value the government would probably pay at least 7. Think of all the misuse possibilities
November 8, 2025 at 1:34 PM
For something of that value the government would probably pay at least 7. Think of all the misuse possibilities
It's not typically lucid, that's why it's distressing for many. Parasomnias and sleep/wake border is tremendous group of variation from the pathological to the normal.
March 2, 2025 at 11:08 PM
It's not typically lucid, that's why it's distressing for many. Parasomnias and sleep/wake border is tremendous group of variation from the pathological to the normal.
Most neurotropics can effect sleep architecture, in those with pre-disposition especially.
February 28, 2025 at 6:34 PM
Most neurotropics can effect sleep architecture, in those with pre-disposition especially.
Hypnogogic hallucinations don't keep consistency, moreoften than not sleep parlysis becomes the typical dream form. I assure you, not a pleasant experience.
February 28, 2025 at 8:02 AM
Hypnogogic hallucinations don't keep consistency, moreoften than not sleep parlysis becomes the typical dream form. I assure you, not a pleasant experience.
It's the lemborexant. It even showed up in trials for small %. It is consistent though and, dose mediated, and likely wouldnt fade as most AEs. Its due to orexin antagonism. Stops upon cessation. You'd be better served by a z-drug.
February 28, 2025 at 7:58 AM
It's the lemborexant. It even showed up in trials for small %. It is consistent though and, dose mediated, and likely wouldnt fade as most AEs. Its due to orexin antagonism. Stops upon cessation. You'd be better served by a z-drug.
Interesting but basis on top of frequent GWAS marker hits is tenuous at best. Sure, guanfacine, and clonidine can bring helpful in some phenotypes but not through reduction of BP alone. Calcium blockers are a very different beast in the brain than alpha blockers.
February 22, 2025 at 3:48 AM
Interesting but basis on top of frequent GWAS marker hits is tenuous at best. Sure, guanfacine, and clonidine can bring helpful in some phenotypes but not through reduction of BP alone. Calcium blockers are a very different beast in the brain than alpha blockers.