Adam Zeman's recent review of mental imagery extremes [1] offers a thorough and balanced synthesis of current perspectives on aphantasia. Among the findings he discusses is a recent connectivity study [2] in which we found that individuals with aphantasia showed typical activation of high-level visual cortices during imagery tasks but reduced functional connectivity between the left fusiform imagery node (FIN) [3] and left frontoparietal regions. Zeman suggests that such alterations in network interactions may provide a promising neural basis for aphantasia.