Upon hyperosmotic stress, the autophagy receptor p62/SQSTM1 rapidly forms biomolecular condensates distinct from stress granules, serving as a platform for autophagic degradation in human cells
Upon hyperosmotic stress, the autophagy receptor p62/SQSTM1 rapidly forms biomolecular condensates distinct from stress granules, serving as a platform for autophagic degradation in human cells