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Upon hyperosmotic stress, the autophagy receptor p62/SQSTM1 rapidly forms biomolecular condensates distinct from stress granules, serving as a platform for autophagic degradation in human cells
Upon hyperosmotic stress, the autophagy receptor p62/SQSTM1 rapidly forms biomolecular condensates distinct from stress granules, serving as a platform for autophagic degradation in human cells
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