Wei Luo
@luoweicool.bsky.social
Assistant Professor of Microbiology and Immunology at Indiana University School of Medicine; Study germinal center and vaccines
It has been an honor to work with such a fantastic team on this project. Special thanks to Dr. Mark Shlomchik, my longtime mentor (and now a collaborator). This is the very first first-author paper for my PhD student, Alex Maldeney, who worked closely with Dr. Wenxia Jiang throughout this work.
August 7, 2025 at 7:07 PM
It has been an honor to work with such a fantastic team on this project. Special thanks to Dr. Mark Shlomchik, my longtime mentor (and now a collaborator). This is the very first first-author paper for my PhD student, Alex Maldeney, who worked closely with Dr. Wenxia Jiang throughout this work.
Actually 8 years ago, forgot it is 2025
July 14, 2025 at 5:42 PM
Actually 8 years ago, forgot it is 2025
Deeply grateful to all the amazing co-authors and trainees who contributed. This was truly a team effort — your hard work made this possible.
July 14, 2025 at 5:23 PM
Deeply grateful to all the amazing co-authors and trainees who contributed. This was truly a team effort — your hard work made this possible.
I initiated this project~5 years ago when I was a postdoc in Dr. Mark Shlomchik’s lab. It’s amazing to bring it full circle — now collaborating as co-corresponding authors after starting my own lab. Very grateful for Dr. Shlomchik’s continued support throughout this journey.
July 14, 2025 at 5:23 PM
I initiated this project~5 years ago when I was a postdoc in Dr. Mark Shlomchik’s lab. It’s amazing to bring it full circle — now collaborating as co-corresponding authors after starting my own lab. Very grateful for Dr. Shlomchik’s continued support throughout this journey.
In GC B cells, high BCL6 expression alters the regulatory hierarchy: BLIMP1 acts upstream, controlling IRF4 expression indirectly through BCL6. This contrasts with naive B cells, where IRF4 functions more upstream of BLIMP1.
July 14, 2025 at 5:23 PM
In GC B cells, high BCL6 expression alters the regulatory hierarchy: BLIMP1 acts upstream, controlling IRF4 expression indirectly through BCL6. This contrasts with naive B cells, where IRF4 functions more upstream of BLIMP1.
Combined signals, such as BCR/CD40 or IL-21R/CD40, can induce BLIMP1 in GC B cells. BLIMP1 limits GC B cell expansion and promotes effector differentiation — not just for plasma cell differentiation but also memory B cell differentiation from GC. A broader role than previously appreciated.
July 14, 2025 at 5:23 PM
Combined signals, such as BCR/CD40 or IL-21R/CD40, can induce BLIMP1 in GC B cells. BLIMP1 limits GC B cell expansion and promotes effector differentiation — not just for plasma cell differentiation but also memory B cell differentiation from GC. A broader role than previously appreciated.
Congratulations, Mark!
March 28, 2025 at 2:48 PM
Congratulations, Mark!
😭 I am so sorry for the POs. They are always very nice to me.
February 17, 2025 at 7:18 PM
😭 I am so sorry for the POs. They are always very nice to me.
Too bad I just submitted one grant with "vaccine" in the title 😂
February 5, 2025 at 3:54 AM
Too bad I just submitted one grant with "vaccine" in the title 😂