kiki 🏳️⚧️🏳️🌈🧬
@kiki-omics.bsky.social
she/they
Lecturer in human genetics at
@exeter.ac.uk | Previously at @ox.ac.uk & @sangerinstitute.bsky.social | Proudly autistic & trans | Latinx
https://experts.exeter.ac.uk/45188-kiki-canogamez
Lecturer in human genetics at
@exeter.ac.uk | Previously at @ox.ac.uk & @sangerinstitute.bsky.social | Proudly autistic & trans | Latinx
https://experts.exeter.ac.uk/45188-kiki-canogamez
A very fulfilling collaboration between @ox.ac.uk @ndm.ox.ac.uk's Centre for Human Genetics, and Ludwig Cancer Research institutes.
Led by Prof Julian Knight. and Associate Prof. Chun-Xiao Song.
(3/3)
Led by Prof Julian Knight. and Associate Prof. Chun-Xiao Song.
(3/3)
February 21, 2025 at 1:30 PM
A very fulfilling collaboration between @ox.ac.uk @ndm.ox.ac.uk's Centre for Human Genetics, and Ludwig Cancer Research institutes.
Led by Prof Julian Knight. and Associate Prof. Chun-Xiao Song.
(3/3)
Led by Prof Julian Knight. and Associate Prof. Chun-Xiao Song.
(3/3)
We evaluate two possible explanations for this increase:
a) cfDNA increases due to excess cell death
b) cfDNA increases because it cannot be cleared efficiently
Our data consistently suggests an impairment in cfDNA clearance in the liver, challenging previous literature.
(2/3)
a) cfDNA increases due to excess cell death
b) cfDNA increases because it cannot be cleared efficiently
Our data consistently suggests an impairment in cfDNA clearance in the liver, challenging previous literature.
(2/3)
February 21, 2025 at 1:30 PM
We evaluate two possible explanations for this increase:
a) cfDNA increases due to excess cell death
b) cfDNA increases because it cannot be cleared efficiently
Our data consistently suggests an impairment in cfDNA clearance in the liver, challenging previous literature.
(2/3)
a) cfDNA increases due to excess cell death
b) cfDNA increases because it cannot be cleared efficiently
Our data consistently suggests an impairment in cfDNA clearance in the liver, challenging previous literature.
(2/3)