Finally, we can look at other associated pathology.
In HF, we will often see other features such as bilateral simple pleural effusions; plus supportive cardiac findings.
In inflammatory/infectious causes, you may see unilateral consolidations, dynamic air bronchograms, shred signs, etc.
Finally, we can look at other associated pathology.
In HF, we will often see other features such as bilateral simple pleural effusions; plus supportive cardiac findings.
In inflammatory/infectious causes, you may see unilateral consolidations, dynamic air bronchograms, shred signs, etc.
B lines arising from pulmonary edema should be bilateral, symmetrical, and in a dependent gradient (ie. most prominent at the bases).
B lines from infectious/inflammatory pathologies, on the other hand, are often asymmetrical, non-gravitational, and may display skipped or spared areas.
B lines arising from pulmonary edema should be bilateral, symmetrical, and in a dependent gradient (ie. most prominent at the bases).
B lines from infectious/inflammatory pathologies, on the other hand, are often asymmetrical, non-gravitational, and may display skipped or spared areas.
In contrast, with infectious/inflammatory pathologies, you will see B lines which are NOT homogenous throughout the interspace.
You may see all the B lines arising from a single point on the pleura; or even A lines throughout part of the interspace.
In contrast, with infectious/inflammatory pathologies, you will see B lines which are NOT homogenous throughout the interspace.
You may see all the B lines arising from a single point on the pleura; or even A lines throughout part of the interspace.
Next, we can look at the distribution of B lines across the interspace.
Pleural fluid fills interlobular septae in a predictable and regular way; which generates B lines that are evenly spaced throughout the whole interspace.
Next, we can look at the distribution of B lines across the interspace.
Pleural fluid fills interlobular septae in a predictable and regular way; which generates B lines that are evenly spaced throughout the whole interspace.
In contrast, B lines arising from an irregular, ragged, interrupted pleural line are more likely to be from infectious/inflammatory pathologies.
(If you want to know WHY this happens - check out the video above!👆)
In contrast, B lines arising from an irregular, ragged, interrupted pleural line are more likely to be from infectious/inflammatory pathologies.
(If you want to know WHY this happens - check out the video above!👆)
And there are several ultrasound features that we can use to tease apart these two types.
First: we can look at the pleural morphology.
B lines arising from a smooth, crisp, uniform pleural line are more likely to be from cardiogenic pulmonary edema.
And there are several ultrasound features that we can use to tease apart these two types.
First: we can look at the pleural morphology.
B lines arising from a smooth, crisp, uniform pleural line are more likely to be from cardiogenic pulmonary edema.
The transhepatic view - sometimes called the rescue view - is another great option for difficult cases ⛑️
This is found in the mid-axillary line in a coronal plane (for the long-axis). From your standard view of the kidney, fan or slide your probe anteriorly until the IVC pops into view.
The transhepatic view - sometimes called the rescue view - is another great option for difficult cases ⛑️
This is found in the mid-axillary line in a coronal plane (for the long-axis). From your standard view of the kidney, fan or slide your probe anteriorly until the IVC pops into view.
Next: don't forget your troubleshooting arsenal 🏹
The IVC can be tough, so it's worth having a couple tricks up your sleeve for challenging cases 🃏
For example - the phased array probe (vs the curvilinear) often nestles better into the subxiphoid space, giving you a better view.
Next: don't forget your troubleshooting arsenal 🏹
The IVC can be tough, so it's worth having a couple tricks up your sleeve for challenging cases 🃏
For example - the phased array probe (vs the curvilinear) often nestles better into the subxiphoid space, giving you a better view.
We also want to be wary of overcalling "IVC collapse" in the long-axis, especially if we're losing the clearly defined borders of the vessel with inspiration 📏
You may be seeing lateral translation of the IVC with respiration - it's moving out of your plane of view - rather than true collapse
We also want to be wary of overcalling "IVC collapse" in the long-axis, especially if we're losing the clearly defined borders of the vessel with inspiration 📏
You may be seeing lateral translation of the IVC with respiration - it's moving out of your plane of view - rather than true collapse
Thirdly: avoid assessing the IVC right at the diaphragm ❌
The diaphragm can either stent the IVC open or "pinch it" during respiration, which is not the same as true collapse.
Train your eye (ideally) a couple cm distal to the hepatic vein
Thirdly: avoid assessing the IVC right at the diaphragm ❌
The diaphragm can either stent the IVC open or "pinch it" during respiration, which is not the same as true collapse.
Train your eye (ideally) a couple cm distal to the hepatic vein
Next: beware the pitfalls of a single view!
This is an axiom that holds true for all of POCUS - always look in more than one view 👍
With the IVC, the short-axis provides a plethora of useful information; including shape/sphericity, which is arguably the most useful parameter of all
Next: beware the pitfalls of a single view!
This is an axiom that holds true for all of POCUS - always look in more than one view 👍
With the IVC, the short-axis provides a plethora of useful information; including shape/sphericity, which is arguably the most useful parameter of all