Congratulations Ora!! We are very happy to have you as a colleague @uni-muenster.de

other community members e.g., studying proteostasis, protein quality mechanism, and retrograde signaling. We hope you enjoy reading our preprint. Thank you.

we find evidence that mistranslation in wild-type plants varies under stress conditions. This suggests that optimization of protein synthesis may improve plant stress tolerance. Moreover, the initial discovery of a hypermistranslating Arabidopsis mutant will benefit

mistranslation was unknown. We isolated a Q➡️E hypermistranslating Arabidopsis mutant and show that plant cells are highly tolerant towards Q➡️E mistranslation in mitochondrial- and plastid-expressed proteins and even incorporate Q➡️E altered peptides into the photosynthetic machinery. Interestingly,

that gene expression works close to perfection, especially protein synthesis is a messy business. Bacteria use protein mistranslation since increased proteome plasticity can aid survival under stress conditions. The relationship of plant cells and their bacterial-type organelles towards protein

And most important: independent of ABA-dependent SnRK2 activity!

We propose that the molecular separation of the DOG1-PP2C module from the regulation of SnRK2 activity keeps seed ABA signaling primed independently of the dormancy level. This allows non-dormant seeds to quickly reinduce ABA responses and arrest germination upon sensing stress.

Thank you so much for this great symposium! Very much enjoyed listening to all the fantastic talks, meetings and for the conference dinner with extra Cologne experience 😃