Dr. Michel Desjardins
@drmicheldesjardins.bsky.social
Research expertise: Antigen presentation, Parkinson's disease and autoimmune diseases.
This is everything you need to know about #longCOVID:
"There are no approved treatments for post-COVID-19 condition (aka #LongCOVID)"
The only way to avoid suffering #LongCOVID is to not get #COVID19.
www.thelancet.com/journals/lan...
"There are no approved treatments for post-COVID-19 condition (aka #LongCOVID)"
The only way to avoid suffering #LongCOVID is to not get #COVID19.
www.thelancet.com/journals/lan...
Targeting the SARS-CoV-2 reservoir in long COVID
There are no approved treatments for post-COVID-19 condition (also known as long COVID),
a debilitating disease state following SARS-CoV-2 infection that is estimated to affect
tens of millions of peo...
www.thelancet.com
February 11, 2025 at 9:38 PM
This is everything you need to know about #longCOVID:
"There are no approved treatments for post-COVID-19 condition (aka #LongCOVID)"
The only way to avoid suffering #LongCOVID is to not get #COVID19.
www.thelancet.com/journals/lan...
"There are no approved treatments for post-COVID-19 condition (aka #LongCOVID)"
The only way to avoid suffering #LongCOVID is to not get #COVID19.
www.thelancet.com/journals/lan...
Reposted by Dr. Michel Desjardins
#Medsky🧪 #IDsky #neurosky #immunosky #publichealth Vascular dysregulation and hypoperfusion are now recognided as central mechanisms for cerebral and muscular disturbance in ME/CFS & #neurocovid
onlinelibrary.wiley.com/doi/full/10....
onlinelibrary.wiley.com/doi/full/10....
Key Pathophysiological Role of Skeletal Muscle Disturbance in Post COVID and Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): Accumulated Evidence
Background Recent studies provide strong evidence for a key role of skeletal muscle pathophysiology in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS). In a 2021 review article on the pa...
onlinelibrary.wiley.com
February 11, 2025 at 5:17 AM
#Medsky🧪 #IDsky #neurosky #immunosky #publichealth Vascular dysregulation and hypoperfusion are now recognided as central mechanisms for cerebral and muscular disturbance in ME/CFS & #neurocovid
onlinelibrary.wiley.com/doi/full/10....
onlinelibrary.wiley.com/doi/full/10....
Periods of relapse and remission of inflammation of the gastrointestinal tract (GI) and IBS association with #Alzheimers and #Parkinsons is not the real concern ...
1/2
pmc.ncbi.nlm.nih.gov/articles/PMC...
1/2
pmc.ncbi.nlm.nih.gov/articles/PMC...
Understanding Gut Feelings: Transformations in Coping With Inflammatory Bowel Disease Among Young Adults
Past studies have revealed a dizzying array of coping techniques employed by persons living with inflammatory bowel disease (IBD). Unfortunately, research has provided little insight into when and why...
pmc.ncbi.nlm.nih.gov
February 11, 2025 at 9:16 PM
Periods of relapse and remission of inflammation of the gastrointestinal tract (GI) and IBS association with #Alzheimers and #Parkinsons is not the real concern ...
1/2
pmc.ncbi.nlm.nih.gov/articles/PMC...
1/2
pmc.ncbi.nlm.nih.gov/articles/PMC...
Evidence that autoimmune mechanisms mediated by T cells play a role in the induction of Parkinson’s disease (PD). Autoimmune mechanisms, including the long-lasting activation of cytotoxic T cells, may explain PD’s extended prodromal period. #Parkinsons #LongCOVID
pubmed.ncbi.nlm.nih.gov/27345367/
pubmed.ncbi.nlm.nih.gov/27345367/
Parkinson's Disease-Related Proteins PINK1 and Parkin Repress Mitochondrial Antigen Presentation - PubMed
Antigen presentation is essential for establishing immune tolerance and for immune responses against infectious disease and cancer. Although antigen presentation can be mediated by autophagy, here we ...
pubmed.ncbi.nlm.nih.gov
February 11, 2025 at 9:07 PM
Evidence that autoimmune mechanisms mediated by T cells play a role in the induction of Parkinson’s disease (PD). Autoimmune mechanisms, including the long-lasting activation of cytotoxic T cells, may explain PD’s extended prodromal period. #Parkinsons #LongCOVID
pubmed.ncbi.nlm.nih.gov/27345367/
pubmed.ncbi.nlm.nih.gov/27345367/