Just submitted my application and working model for my NIH F31 for #BioRenderForGrants! This model has implications not only in alcohol research, but also in various other fields. If we can force cells to “reroute” to a different metabolic pathway, we may be able to restore effector function.

It has also been published that AhR and NF-kB can interact and compete signaling-wise. I would be interested to know more about that in this case, especially in cases where subunits in both pathways complex with one another.

Had some really good conversations at the poster session today at #RSA_ISBRA2025! Thank you to everyone who came and chatted with me! 🥂

Gut Pathogens is a part of Springer Nature, whose policy states that AI cannot be considered an author, and use of a learning language model should be stated in the paper. In this paper the use of AI is not mentioned anywhere.

AI-generated images are not permitted, with some exceptions.

This figure is what really gave it away to me. Words like “bloodstroom/bloedstream” “aimmune” and “Erysipelotrichacee”, multiple misleading labels of the vagus nerve and the weird labeling of lymphatics gives it away.

This paper was published in Gut Pathogens a little over a week ago.

I am eternally grateful for the opportunity to close out our GRC conference last night. The alcohol research field is full of incredible individuals doing cutting edge research and I am so very lucky and blessed not only to know these people but give them insight on my own endeavors. Until 2027 ❤️🥂