Human cysticercosis results from ingesting microscopic Taenia solium eggs shed by a tapeworm carrier, forming cystic larvae (ie, cysticerci) in tissues. These larvae normally encyst in the pig, the natural intermediate host. Upon consumption by humans, they develop into a new intestinal tapeworm. Affecting the human brain, neurocysticercosis is responsible for a third of epilepsy cases in endemic tropical and subtropical regions.1,2

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