Gaia Ghirardi
@gaiaghirardi.bsky.social
Sociologist for children and social equality.
Postdoc at University of Bologna (GENPOP) & previously PhD at the EUI.
https://gaiaghirardi.github.io
Postdoc at University of Bologna (GENPOP) & previously PhD at the EUI.
https://gaiaghirardi.github.io
Also, be sure not to miss the *extensive supplementary materials*, which include many detailed sections 🤓
journals.sagepub.com/doi/10.1177/...
5/5
journals.sagepub.com/doi/10.1177/...
5/5
Sage Journals: Discover world-class research
Subscription and open access journals from Sage, the world's leading independent academic publisher.
journals.sagepub.com
November 6, 2025 at 1:12 PM
Also, be sure not to miss the *extensive supplementary materials*, which include many detailed sections 🤓
journals.sagepub.com/doi/10.1177/...
5/5
journals.sagepub.com/doi/10.1177/...
5/5
Main takeaways:
1) ECEC is the most effective strategy to reduce social disparities, positively affecting low-SES children
2) Exclusive parental care and family day-care may increase social disparities, benefiting high-SES children while negatively affecting low-SES
4/5
1) ECEC is the most effective strategy to reduce social disparities, positively affecting low-SES children
2) Exclusive parental care and family day-care may increase social disparities, benefiting high-SES children while negatively affecting low-SES
4/5
November 6, 2025 at 1:12 PM
Main takeaways:
1) ECEC is the most effective strategy to reduce social disparities, positively affecting low-SES children
2) Exclusive parental care and family day-care may increase social disparities, benefiting high-SES children while negatively affecting low-SES
4/5
1) ECEC is the most effective strategy to reduce social disparities, positively affecting low-SES children
2) Exclusive parental care and family day-care may increase social disparities, benefiting high-SES children while negatively affecting low-SES
4/5
Applying this novel decomposition approach, we estimate the overall relevance of each childcare arrangement, the importance of specific channels impacting social disparities (prevalence, effect, and selection)
projecteuclid.org/journals/ann...
3/5
projecteuclid.org/journals/ann...
3/5
Nonparametric causal decomposition of group disparities
We introduce a new nonparametric causal decomposition approach that identifies the mechanisms by which a treatment variable contributes to a group-based outcome disparity. Our approach distinguishes three mechanisms: group differences in: (1) treatment prevalence, (2) average treatment effects, and (3) selection into treatment based on individual-level treatment effects. Our approach reformulates classic Kitagawa–Blinder–Oaxaca decompositions in causal and nonparametric terms, complements causal mediation analysis by explaining group disparities instead of group effects, and isolates conceptually distinct mechanisms conflated in recent random equalization decompositions. In contrast to all prior approaches, our framework uniquely identifies differential selection into treatment as a novel disparity-generating mechanism. Our approach can be used for both the retrospective causal explanation of disparities and the prospective planning of interventions to change disparities. We present both an unconditional and a conditional decomposition, where the latter quantifies the contributions of the treatment within levels of certain covariates. We develop nonparametric estimators that are n-consistent, asymptotically normal, semiparametrically efficient, and multiply robust. We apply our approach to analyze the mechanisms by which college graduation causally contributes to intergenerational income persistence (the disparity in adult income between the children of high- vs. low-income parents). Empirically, we demonstrate a previously undiscovered role played by the new selection component in intergenerational income persistence.
projecteuclid.org
November 6, 2025 at 1:12 PM
Applying this novel decomposition approach, we estimate the overall relevance of each childcare arrangement, the importance of specific channels impacting social disparities (prevalence, effect, and selection)
projecteuclid.org/journals/ann...
3/5
projecteuclid.org/journals/ann...
3/5
Using the newborn sample of the NEPS data, we look at the role of ECEC, family day care, grandparental care, and exclusive parental care on children’s competencies in mathematics and vocabulary (4- and 5-year-olds)
2/5
2/5
November 6, 2025 at 1:12 PM
Using the newborn sample of the NEPS data, we look at the role of ECEC, family day care, grandparental care, and exclusive parental care on children’s competencies in mathematics and vocabulary (4- and 5-year-olds)
2/5
2/5
So far: some suggestions of publication bias but it seems driven by studies looking at cognitive skills 🧐
October 8, 2025 at 12:13 PM
So far: some suggestions of publication bias but it seems driven by studies looking at cognitive skills 🧐
Pure speculation, but maybe the trauma of the false-positive candidate gene papers made us more likely to publish null GxE results using polygenic scores. But I might just be imagining it 😊
October 8, 2025 at 10:45 AM
Pure speculation, but maybe the trauma of the false-positive candidate gene papers made us more likely to publish null GxE results using polygenic scores. But I might just be imagining it 😊
😅
Btw @astabreinholt.bsky.social and I are doing a meta-analysis on GxSES studies on edu outcomes assessing publication bias and so on - but it’s still in progress 😊
Btw @astabreinholt.bsky.social and I are doing a meta-analysis on GxSES studies on edu outcomes assessing publication bias and so on - but it’s still in progress 😊
October 8, 2025 at 10:06 AM
😅
Btw @astabreinholt.bsky.social and I are doing a meta-analysis on GxSES studies on edu outcomes assessing publication bias and so on - but it’s still in progress 😊
Btw @astabreinholt.bsky.social and I are doing a meta-analysis on GxSES studies on edu outcomes assessing publication bias and so on - but it’s still in progress 😊
This is the last paper of my PhD thesis (🥹) and I want to thank those who provided feedback on this final chapter, among them: @fabriberna.bsky.social, @marcocozzani.bsky.social, @melindacmills.bsky.social, @nicolabarban.bsky.social, @juhoharkonen.bsky.social, and the SSM editors and reviewers 🙏🏼
5/5
5/5
September 17, 2025 at 10:56 AM
This is the last paper of my PhD thesis (🥹) and I want to thank those who provided feedback on this final chapter, among them: @fabriberna.bsky.social, @marcocozzani.bsky.social, @melindacmills.bsky.social, @nicolabarban.bsky.social, @juhoharkonen.bsky.social, and the SSM editors and reviewers 🙏🏼
5/5
5/5
Contributing to research on health inequalities and sociogenomics, I find:
1) PGI BMI is more predictive of high levels of BMI among individuals from *low-SES families*
2) This pattern is weakened over the life course but *persists* among individuals with high BMI levels
4/5
1) PGI BMI is more predictive of high levels of BMI among individuals from *low-SES families*
2) This pattern is weakened over the life course but *persists* among individuals with high BMI levels
4/5
September 17, 2025 at 10:56 AM
Contributing to research on health inequalities and sociogenomics, I find:
1) PGI BMI is more predictive of high levels of BMI among individuals from *low-SES families*
2) This pattern is weakened over the life course but *persists* among individuals with high BMI levels
4/5
1) PGI BMI is more predictive of high levels of BMI among individuals from *low-SES families*
2) This pattern is weakened over the life course but *persists* among individuals with high BMI levels
4/5
This work tests the expectation from the compensatory advantage and diathesis-stress theories by using the Add Health data. Specifically, it assesses whether the association between a PGI BMI and BMI at different life stages (ages 16, 22, 28, and 37) is shaped by family socioeconomic status.
3/5
3/5
September 17, 2025 at 10:56 AM
This work tests the expectation from the compensatory advantage and diathesis-stress theories by using the Add Health data. Specifically, it assesses whether the association between a PGI BMI and BMI at different life stages (ages 16, 22, 28, and 37) is shaped by family socioeconomic status.
3/5
3/5
I study the genotype-family SES interaction on obesity and overweight across the life-course.
I ask whether the genetic propensity for high BMI matters more in explaining overweight and obesity among individuals from low or high-SES families? And if so, does this interaction vary by age?
2/5
I ask whether the genetic propensity for high BMI matters more in explaining overweight and obesity among individuals from low or high-SES families? And if so, does this interaction vary by age?
2/5
September 17, 2025 at 10:56 AM
I study the genotype-family SES interaction on obesity and overweight across the life-course.
I ask whether the genetic propensity for high BMI matters more in explaining overweight and obesity among individuals from low or high-SES families? And if so, does this interaction vary by age?
2/5
I ask whether the genetic propensity for high BMI matters more in explaining overweight and obesity among individuals from low or high-SES families? And if so, does this interaction vary by age?
2/5
This work tests the expectation from the compensatory advantage and diathesis-stress theories by using the Add Health data. Specifically, it assesses whether the association between a PGI BMI and BMI at different life stages (ages 16, 22, 28, and 37) is shaped by family socioeconomic status.
3/5
3/5
September 17, 2025 at 10:50 AM
This work tests the expectation from the compensatory advantage and diathesis-stress theories by using the Add Health data. Specifically, it assesses whether the association between a PGI BMI and BMI at different life stages (ages 16, 22, 28, and 37) is shaped by family socioeconomic status.
3/5
3/5
I study the genotype-family SES interaction on obesity and overweight across the life-course.
I ask whether the genetic propensity for high BMI matters more in explaining overweight and obesity among individuals from low or high-SES families? And if so, does this interaction vary by age?
2/5
I ask whether the genetic propensity for high BMI matters more in explaining overweight and obesity among individuals from low or high-SES families? And if so, does this interaction vary by age?
2/5
September 17, 2025 at 10:50 AM
I study the genotype-family SES interaction on obesity and overweight across the life-course.
I ask whether the genetic propensity for high BMI matters more in explaining overweight and obesity among individuals from low or high-SES families? And if so, does this interaction vary by age?
2/5
I ask whether the genetic propensity for high BMI matters more in explaining overweight and obesity among individuals from low or high-SES families? And if so, does this interaction vary by age?
2/5