Eddie Rashan
@edreesrashan.bsky.social
Lipid metabolism biochemist. Postdoc in the Vander Heiden Lab at MIT | he/him | ORCID: https://orcid.org/0000-0002-3882-6655
Awesome work!! Congratulations!
October 30, 2025 at 11:30 AM
Awesome work!! Congratulations!
Thank you, CJ! Hope you and your lab are well.
June 23, 2025 at 12:51 PM
Thank you, CJ! Hope you and your lab are well.
It was a privilege to work on a project that allow us to span multiple disciplines and reveal new fundamental biology. Thank you to our funding support, incl. from the NIH, @hhmi.org , and institutional support @washumedicine.bsky.social @uwbiochem.bsky.social @morgridgeinstitute.bsky.social
June 22, 2025 at 11:41 PM
It was a privilege to work on a project that allow us to span multiple disciplines and reveal new fundamental biology. Thank you to our funding support, incl. from the NIH, @hhmi.org , and institutional support @washumedicine.bsky.social @uwbiochem.bsky.social @morgridgeinstitute.bsky.social
We hope our findings will spur future studies to clarify how these enzymes and the regulation of 4-HAs underlie human health and disease. There is much to learn about how 4-HAs contribute to whole body metabolic flexibility and their tissue- and organelle-specific functions!
June 22, 2025 at 11:38 PM
We hope our findings will spur future studies to clarify how these enzymes and the regulation of 4-HAs underlie human health and disease. There is much to learn about how 4-HAs contribute to whole body metabolic flexibility and their tissue- and organelle-specific functions!
Lastly, we wanted to determine how dysfunctional 4-HA catabolism affects physiology and generated ACAD11 KO mice. Disruption of ACAD11 in vivo leads to accumulation in plasma 4-HAs, higher susceptibility to diet-induced fat gain, and dysfunctional adipogenesis. 🧐
June 22, 2025 at 11:36 PM
Lastly, we wanted to determine how dysfunctional 4-HA catabolism affects physiology and generated ACAD11 KO mice. Disruption of ACAD11 in vivo leads to accumulation in plasma 4-HAs, higher susceptibility to diet-induced fat gain, and dysfunctional adipogenesis. 🧐