Danny
@dannybar.bsky.social
Molecular biology of aging at Tel Aviv University.
DanielBar.eth
DanielBar.eth
information loss in the liver. Thanks to the
Goldstein lab that provided much of the data and the ChIP-seq analysis. biorxiv.org/content/10.1...
P.S. the preprint isn't really new, but you aren't following me on twitter, so it's new for you.
Goldstein lab that provided much of the data and the ChIP-seq analysis. biorxiv.org/content/10.1...
P.S. the preprint isn't really new, but you aren't following me on twitter, so it's new for you.
June 14, 2025 at 7:04 PM
information loss in the liver. Thanks to the
Goldstein lab that provided much of the data and the ChIP-seq analysis. biorxiv.org/content/10.1...
P.S. the preprint isn't really new, but you aren't following me on twitter, so it's new for you.
Goldstein lab that provided much of the data and the ChIP-seq analysis. biorxiv.org/content/10.1...
P.S. the preprint isn't really new, but you aren't following me on twitter, so it's new for you.
and some diseases. Here, we show that these sites causally contribute to aging: both pathogenic SNPs and causal epigenetic clocks (DamAge) are enriched for these sites. Information loss is also reversible: in mice, repeated fasting (but not a single fast) reverses this 2/3
June 14, 2025 at 7:04 PM
and some diseases. Here, we show that these sites causally contribute to aging: both pathogenic SNPs and causal epigenetic clocks (DamAge) are enriched for these sites. Information loss is also reversible: in mice, repeated fasting (but not a single fast) reverses this 2/3
Fun fact - this was the inspiration for the review process in journal XXX.
June 12, 2025 at 9:34 AM
Fun fact - this was the inspiration for the review process in journal XXX.