Daichi Inoue
@daichi-inoue.bsky.social
Hematologist, Science and Patients Lover. Professor, Dep. of Cancer Pathology, Osaka University🇯🇵
🩸🦴🧬💊🔬🌎⛰️🏃♂️🐭⛩️🇺🇸🗾🫂
MD@Kyoto Univ. → PhD@Univ. of Tokyo. → Pos-doc@Sloan Kettering (NYC) → Junior PI@FBRI (Kobe) → Professor@Osaka.
🩸🦴🧬💊🔬🌎⛰️🏃♂️🐭⛩️🇺🇸🗾🫂
MD@Kyoto Univ. → PhD@Univ. of Tokyo. → Pos-doc@Sloan Kettering (NYC) → Junior PI@FBRI (Kobe) → Professor@Osaka.
This work would not have been possible without the unwavering dedication of Ph.D. candidate Dr. Aoyama and Assistant Prof. Yamazaki. We are also sincerely grateful for the fruitful collaboration with Japanese societies and co-authors.
February 6, 2025 at 6:48 AM
This work would not have been possible without the unwavering dedication of Ph.D. candidate Dr. Aoyama and Assistant Prof. Yamazaki. We are also sincerely grateful for the fruitful collaboration with Japanese societies and co-authors.
(3) Under such oxidative stress, B cell progenitors acquire myeloid characteristics.
(4) Dietary interventions can modulate these effects.
Here is the link for news release
www.eurekalert.org/news-release...
(4) Dietary interventions can modulate these effects.
Here is the link for news release
www.eurekalert.org/news-release...
Selenoproteins: The fountain of youth?
Researchers from Japan find that certain antioxidant enzymes, called selenoproteins, significantly contribute to fighting cell aging. The team used a gene knockout mouse model to help them study the e...
www.eurekalert.org
February 6, 2025 at 6:48 AM
(3) Under such oxidative stress, B cell progenitors acquire myeloid characteristics.
(4) Dietary interventions can modulate these effects.
Here is the link for news release
www.eurekalert.org/news-release...
(4) Dietary interventions can modulate these effects.
Here is the link for news release
www.eurekalert.org/news-release...
Through extensive analysis of the KO animal model for selenocysteine tRNA and human data, we demonstrate that
(1) Disrupted selenoprotein synthesis mimics an aged phenotype.
(2) The accumulation of lipid peroxidation accelerated by aging occurs in specific cellular contexts and lineages.
(1) Disrupted selenoprotein synthesis mimics an aged phenotype.
(2) The accumulation of lipid peroxidation accelerated by aging occurs in specific cellular contexts and lineages.
February 6, 2025 at 6:48 AM
Through extensive analysis of the KO animal model for selenocysteine tRNA and human data, we demonstrate that
(1) Disrupted selenoprotein synthesis mimics an aged phenotype.
(2) The accumulation of lipid peroxidation accelerated by aging occurs in specific cellular contexts and lineages.
(1) Disrupted selenoprotein synthesis mimics an aged phenotype.
(2) The accumulation of lipid peroxidation accelerated by aging occurs in specific cellular contexts and lineages.