Asaf (Klaf) Weisman
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asafklaf.bsky.social
Asaf (Klaf) Weisman
@asafklaf.bsky.social
PhD candidate at Tel-Aviv University. I am a researcher of the spine and people experiencing pain. I am also an MSK Physiotherapist. Follow me for the neuroscience of pain, spine, and health issues.

Blog: https://painlosophy.wordpress.com/
Potential and actual tissue damage are encoded the same. Both qualify as nociception. It is an ingenious evolutionary adaptation that is meant to protect us way before actual damage occurs.
October 16, 2025 at 11:27 AM
Pinch yourself slightly hard and you will likely feel pain due to potential tissue damage.
October 16, 2025 at 10:25 AM
They found a population of noxious heat and chemical irritation processing neurons - not “pain” and not “itch specific neurons”
April 30, 2025 at 1:56 PM
The obvious questions are: how did they get those mice to report they were experiencing pain? And since when pain is being processed anywhere in the brain when pain is a response and the outcome of all processing?
March 24, 2025 at 2:26 PM
Fibromyalgia is a rabbit hole by itself. What have you learned from your quest?
February 16, 2025 at 8:59 AM
It just means that the mode of activation is not nociceptive nor neuropathic. Unfortunately, the term nociplastic pain has been hijacked by those with vested interests to sell and promote psychological treatments for those with “non structural pain. Do not cooperate with this trend!
January 22, 2025 at 1:27 PM
This activation can occur due to three mechanisms: nociceptive, neuropathic and nociplastic. It is now known that activation of nociception can occur independently and without observed tissue damage (nociplastic). This does not make nociplastic “non structural pain”
January 22, 2025 at 1:27 PM