Marta Monguió Tortajada
@mmonguio.bsky.social
Innate immunity & extracellular vesicles in unwanted inflammation & disease.
Postdoc at @rebsamenlab.bsky.social | UNIL @dib-unil.bsky.social
Badalona-Lausanne, she/her
Postdoc at @rebsamenlab.bsky.social | UNIL @dib-unil.bsky.social
Badalona-Lausanne, she/her
Check out the last publication from our lab!
It was a pretty nice Birthday present to the great PhD student @lbernaleau.bsky.social leading this well-laced story on TLR biogenesis: CCDC134 is key for Gp96 function!
It was a pretty nice Birthday present to the great PhD student @lbernaleau.bsky.social leading this well-laced story on TLR biogenesis: CCDC134 is key for Gp96 function!
Very happy to share our latest work just published in @jexpmed.bsky.social! #Firstpost
rupress.org/jem/article/...
Our amazing PhD student @lbernaleau.bsky.social shows that CCDC134 controls TLR biogenesis through the ER-chaperone Gp96. #ImmunoSky #innateimmunity #inflammation
Quick summary here:
rupress.org/jem/article/...
Our amazing PhD student @lbernaleau.bsky.social shows that CCDC134 controls TLR biogenesis through the ER-chaperone Gp96. #ImmunoSky #innateimmunity #inflammation
Quick summary here:
CCDC134 controls TLR biogenesis through the ER chaperone Gp96 | Journal of Experimental Medicine | Rockefeller University Press
A loss-of-function genetic screen identifies the ER-resident protein CCDC134 as an essential regulator of Toll-like receptor (TLR) responses. CCDC134 binds
rupress.org
December 10, 2024 at 9:26 PM
Check out the last publication from our lab!
It was a pretty nice Birthday present to the great PhD student @lbernaleau.bsky.social leading this well-laced story on TLR biogenesis: CCDC134 is key for Gp96 function!
It was a pretty nice Birthday present to the great PhD student @lbernaleau.bsky.social leading this well-laced story on TLR biogenesis: CCDC134 is key for Gp96 function!