Katelyn Connelly, Ph.D.
@kconnelly92.bsky.social
K99/R00 Postdoc
Molecular mechanisms of inherited risk | pancreatic cancer| chromatin | epigenetics | iPSC organoids | functional genomics | molecular mechanisms of gene-environment interplay
Views are my own
Molecular mechanisms of inherited risk | pancreatic cancer| chromatin | epigenetics | iPSC organoids | functional genomics | molecular mechanisms of gene-environment interplay
Views are my own
It’s hard not being able to attend ASHG this year but following all of the happenings through the #ASHG25 posts & looking forward to next year 🧬
October 16, 2025 at 3:10 PM
It’s hard not being able to attend ASHG this year but following all of the happenings through the #ASHG25 posts & looking forward to next year 🧬
Congratulations to my labmates @junzhong.bsky.social and Aidan for their new publication on non-coding somatic mutations in PDAC! I’m honored to have been a part of the work & @amundadottir.bsky.social team.
Congratulations to the @amundadottir.bsky.social team for their #Gut paper reporting a large-scale multiomic analysis of pancreatic ductal adenocarcinoma. Delighted to contribute along with @adwellspenn.bsky.social. @chopresearch.bsky.social @penngenetics.bsky.social
gut.bmj.com/content/earl...
gut.bmj.com/content/earl...
Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene for pancreatic ductal adenocarcinoma
Background The identification and characterisation of somatic cancer driver mutations in the non-coding genome remains challenging.
Objective To broadly characterise non-coding driver mutations for p...
gut.bmj.com
October 10, 2025 at 5:41 PM
Congratulations to my labmates @junzhong.bsky.social and Aidan for their new publication on non-coding somatic mutations in PDAC! I’m honored to have been a part of the work & @amundadottir.bsky.social team.
I'm excited to share that our functional characterization of the 1p36.33 pancreatic cancer risk locus, is now live in Nature Communications. @amundadottir.bsky.social
www.nature.com/articles/s41...
www.nature.com/articles/s41...
Allelic effects on KLHL17 expression underlie a pancreatic cancer genome-wide association signal at chr1p36.33 - Nature Communications
Allele-preferential transcription factor binding can influence pancreatic ductal adenocarcinoma risk loci function. Here, the authors show allele-specific JunB and JunD binding at chr1p36.33 and propo...
www.nature.com
May 2, 2025 at 5:04 PM
I'm excited to share that our functional characterization of the 1p36.33 pancreatic cancer risk locus, is now live in Nature Communications. @amundadottir.bsky.social
www.nature.com/articles/s41...
www.nature.com/articles/s41...