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CONCLUSIONS: In patients with ischemic stroke, the severity of myocardial injury in general but not the presence of acute myocardial injury at time of stroke is associated with cognitive impairment.

The amyloid cascade hypothesis, proposed over 30 years ago, places amyloid-β (Aβ) at the center of Alzheimer's disease (AD) pathogenesis. Though controversial, recent clinical successes with Aβ-targeting therapies have reinforced its importance. However, these treatments have shown only modest clinical benefits in line with a two-stage AD progression: an early phase driven by Aβ-seed and a later phase that progresses at least partly independently of Aβ. Evidence of Aβ seed transmission in humans raises both therapeutic potential and biosafety concerns. This review explores current understanding of Aβ seeds, including challenges in studying such seeds, model systems to study Aβ seeds, and biosafety issues when working with Aβ seeds.

cyCONDOR 0.3.1 changelog Added function condor_info to easily explore the content of a condor object. Solved Issue #31, color pallette is reversed in plot_marker_ridgeplot() function. Solved Issue #30, the pseudotime function can now use a prefix argument. Improved documentation regarding the use of different co-factors when using the archsin data transformation. Solved Issue #33, the plot_marker_dotplot function have now an alpha argument to control the transparency of the dots. Solved Issue #34, the plot_marker_ridgeplot now have a title argument. Solved Issue #19, if an empty fcs or csv file is provided the prep_fcd function will stop and point to the corrupted file. Added function order_param to allow manual ordering of an annotation or clustering variable. Added prefix argument to astir functions.

Socially inappropriate behavior accompanies and modulates delinquency across the lifespan. In contrast to young people, the emergence of such traits among older individuals could indicate incipient neurodegenerative disease. Before developing a neurocognitive disorder, subtle behavioral changes may reflect a disintegration of neural networks involved in impulse control or social cognition. Whereas psychiatric evaluation often considers a comprehensive cognitive assessment, unremarkable results may discourage clinicians from recognizing brain disease underlying behavioral disturbance. We first provide an overview of its manifestations and neural correlates and the interrelations with mild cognitive impairment (MCI) before demonstrating how to investigate and diagnose mild behavioral impairment (MBI). Finally, we show how to appreciate MBI in geriatric forensic psychiatry. Haussmann, Robert; Lange, Jan; Donix, Markus

The amyloid cascade hypothesis, proposed over 30 years ago, places amyloid-β (Aβ) at the center of Alzheimer's disease (AD) pathogenesis. Though controversial, recent clinical successes with Aβ-targeting therapies have reinforced its importance. However, these treatments have shown only modest clinical benefits in line with a two-stage AD progression: an early phase driven by Aβ-seed and a later phase that progresses at least partly independently of Aβ. Evidence of Aβ seed transmission in humans raises both therapeutic potential and biosafety concerns. This review explores current understanding of Aβ seeds, including challenges in studying such seeds, model systems to study Aβ seeds, and biosafety issues when working with Aβ seeds.

Socially inappropriate behavior accompanies and modulates delinquency across the lifespan. In contrast to young people, the emergence of such traits among older individuals could indicate incipient neurodegenerative disease. Before developing a neurocognitive disorder, subtle behavioral changes may reflect a disintegration of neural networks involved in impulse control or social cognition. Whereas psychiatric evaluation often considers a comprehensive cognitive assessment, unremarkable results may discourage clinicians from recognizing brain disease underlying behavioral disturbance. We first provide an overview of its manifestations and neural correlates and the interrelations with mild cognitive impairment (MCI) before demonstrating how to investigate and diagnose mild behavioral impairment (MBI). Finally, we show how to appreciate MBI in geriatric forensic psychiatry.